Description
Product Description:
Semax is a synthetic peptide derived from a fragment of adrenocorticotropic hormone (ACTH 4-10), modified to enhance stability and activity in research settings. It has been widely studied for its interaction with neurotrophic factors, neurotransmitter systems, and central nervous system signaling pathways.
In preclinical and experimental models, Semax has been investigated for its role in brain-derived neurotrophic factor (BDNF) regulation, dopamine and serotonin pathway modulation, and peptide-mediated neurochemical signaling. These properties make it a valuable compound for research focused on neurobiology, cognitive processes, and CNS pathway dynamics.
Semax is commonly utilized in in vitro and laboratory-based research models examining neuropeptide activity, receptor interactions, and neuronal signaling mechanisms.
Key Features:
Chemical Name: Semax (ACTH 4-7 Pro-Gly-Pro)
Molecular Formula: C₃₇H₅₁N₉O₁₀S
CAS Number: 80714-61-0
Amino Acid Sequence: Met-Glu-His-Phe-Pro-Gly-Pro
Physical Form: Lyophilized powder
Compound Class: Synthetic peptide / ACTH analog
Purity: ≥98% (HPLC)
Storage Conditions: Store at 36–46°F (2–8°C) in a dry, light-protected environment.
Common Research Pairings:
Semax is sometimes studied alongside other compounds in neurobiological and peptide signaling research, including:
- Selank – investigated in complementary neuropeptide pathway studies
- DSIP – studied in neuroendocrine and CNS signaling models
- Cerebrolysin – explored in neurotrophic and neuronal pathway research
- Pinealon – examined in gene expression and neurobiological studies
These combinations are referenced strictly within controlled research environments to evaluate complementary biological mechanisms.
Research References:
- Ashmarin, I. P. et al. (1997). Semax and its effects on CNS signaling and neuropeptide systems. Neuroscience and Behavioral Physiology.
- Zolotarev, Y. A. et al. (2004). Semax and modulation of BDNF and neurotransmitter pathways. Bulletin of Experimental Biology and Medicine.
- Gudasheva, T. A. et al. (2016). Neuropeptide regulation and mechanisms of Semax activity. Neurochemical Journal.
(References provided for informational and research context only.)
Important Notice:
This product is intended for research use only. It is not for human or veterinary use. It is not intended to diagnose, treat, cure, or prevent any disease. Use is restricted to qualified professionals in controlled laboratory environments.






